The beauty of the complexity of our biological evolution with microbes and further evidence that supports “Acquiring Genomes: A Theory of the Origin of Species” by Lynn Margulis and Dorion Sagan is still occurring today. I so wish she were still alive because I would have immediately sent her this remarkable scientific finding. It is also further evidence of how perverted capitalism is towards undermining infant development and destroying their health that current hospital practices sabotage even mothers who choose to nurse their own babies. Formula is a Big Pharma product that has been sold to mothers all over the globe. They are not taught how the milk their own bodies produce are instrumental in calibrating the immune system and brain development of their infants.
I Contain Multitudes: The Microbes Within Us and a Grander View of Life by Ed Yong book excerpt.
“It is unclear why human breast milk stands out among that of other mammals. It has five times as many types of H.M.O.s as cow’s milk, and several hundred times the quantity. Even chimp milk is impoverished compared with ours. Mills suggests a couple of possible explanations for this difference. One involves our brains, which are famously large for a primate of our size, and which grow incredibly quickly during our first year of life. This fast growth partly depends on a nutrient called sialic acid, which also happens to be one of the chemicals that B. infantis releases while it eats H.M.O.s. It is possible that, by keeping this bacterium well fed, mothers can raise brainier babies. This might explain why, among monkeys and apes, social species have more milk oligosaccharides than solitary ones, and a greater range of them to boot. Living in larger groups requires remembering more social ties, managing more friendships, and manipulating more rivals. Many scientists believe that these demands drove the evolution of primate intelligence; perhaps they also fueled the diversity of H.M.O.s.
An alternative idea involves diseases. In a group setting, pathogens can easily bounce from one host to another, so animals need better ways of protecting themselves. H.M.O.s provide one such defense. When a pathogen infects our guts, it almost always begins by latching onto glycans—sugar molecules—on the surfaces of our intestinal cells. But H.M.O.s bear a striking resemblance to these glycans, so pathogens sometimes stick to them instead. They act as decoys, drawing fire away from a baby’s own cells. They can block a roll call of gut villains, including Salmonella; Listeria; Vibrio cholerae, the culprit behind cholera; Campylobacter jejuni, the most common cause of bacterial diarrhea; Entamoeba histolytica, a voracious amoeba that causes dysentery and kills a hundred thousand people every year; and many virulent strains of E. coli. H.M.O.s may even be able to obstruct H.I.V., which might explain why more than half of infants who suckle from infected mothers don’t get infected, despite drinking virus-loaded milk for months. Every time scientists have pitted a pathogen against cultured cells in the presence of H.M.O.s, the cells have come out smiling…
scientists have identified more than two hundred human milk oligosaccharides, or H.M.O.s. They are the third-most plentiful ingredient in human milk, after lactose and fats, and their structure ought to make them a rich source of energy for growing babies—but babies cannot digest them. When German first learned this, he was gobsmacked. Why would a mother expend so much energy manufacturing these complicated chemicals if they were apparently useless to her child? Why hasn’t natural selection put its foot down on such a wasteful practice? Here’s a clue: H.M.O.s pass through the stomach and the small intestine unharmed, landing in the large intestine, where most of our bacteria live. What if they aren’t food for babies at all? What if they are food for microbes?…
In 2006, the team found that the sugars selectively nourish one subspecies, Bifidobacterium longum infantis. As long as you provide B. infantis with H.M.O.s, it will outcompete any other gut bacterium. A closely related subspecies, B. longum longum, grows weakly on the same sugars, and the ironically named B. lactis, a common fixture of probiotic yogurts, doesn’t grow at all. Another probiotic mainstay, B. bifidum, does slightly better, but is a fussy, messy eater. It breaks down a few H.M.O.s and takes in the pieces it likes. By contrast, B. infantis devours every last crumb using a cluster of thirty genes—a comprehensive cutlery set for eating H.M.O.s. No other Bif has this genetic cluster; it is unique to B. infantis. Human milk has evolved to nourish the microbe, and it, in turn, has evolved into a consummate H.M.O.vore. Unsurprisingly, it is often the dominant microbe in the guts of breast-fed infants. B. infantis earns its keep. As it digests H.M.O.s, it releases short-chain fatty acids, which feed an infant’s gut cells. Through direct contact, B. infantis also encourages gut cells to make adhesive proteins that seal the gaps between them, keeping microbes out of the bloodstream, and anti-inflammatory molecules that calibrate the immune system. These changes only happen when B. infantis feeds on H.M.O.s; if it gets lactose instead, it survives but doesn’t engage in any repartee with the baby’s cells. In other words, the microbe’s full beneficial potential is unlocked only when it feeds on breast milk.” (Pages 92 – 96)
https://www.newyorker.com/tech/elements/breast-feeding-the-microbiome
https://www.goodreads.com/book/show/27213168-i-contain-multitudes
In other words, your baby’s immune system and brain will never fully develop because they never received your breast milk and they will be more prone to diseases throughout their lives. The same ruling class industrialists who created products off of their munitions technologies that have the precision of sniper fire on fetal development want women to feed their babies their formulas instead because it maximizes their
profits. (Should note that some of their products even destroy mammary development so some women are not even capable of nursing their babies. It’s a win win situation for them…)
“The study showed that the exclusively breastfed group had the fastest growth in myelinated white matter of the three groups, with the increase in white matter volume becoming substantial by age 2. The group fed both breastmilk and formula had more growth than the exclusively formula-fed group, but less than the breastmilk-only group.”
“We’re finding the difference [in white matter growth] is on the order of 20 to 30 percent, comparing the breastfed and the non-breastfed kids,” said Deoni. “I think it’s astounding that you could have that much difference so early.”
https://www.sciencedaily.com/releases/2013/06/130606141048.htm
Perchlorate (rocket fuel) contaminates all formula brands too
CDC found detectable levels of perchlorate in all 2820 urine samples tested, indicating widespread human exposure to perchlorate.”We found significantly higher levels of urinary perchlorate in children compared with adolescents and adults.”
“Perchlorate is commonly found in the environment and can impair thyroid function at pharmacological doses. As a result of the potential for widespread human exposure to this biologically active chemical, we assessed perchlorate exposure in a nationally representative population”
Thank you Wayback machine… (You can read the CDC removed report here.)
https://web.archive.org/web/20091129164121/https://www.cdc.gov/exposurereport/perchlorate1.htm
The CDC tested all infant powdered formulas and found perchlorate contamination in all of the brands. Here’s the CDC study.
Perchlorate exposure from infant formula and comparisons with the perchlorate reference dose.
J Expo Sci Environ Epidemiol. 2010 May;20(3):281-7. doi: 10.1038/jes.2009.18. Epub 2009 Mar 18.
Schier JG1, Wolkin AF, Valentin-Blasini L, Belson MG, Kieszak SM, Rubin CS, Blount BC.
Abstract
Perchlorate exposure may be higher in infants compared with older persons, due to diet (infant formula) and body weight versus intake considerations. Our primary objective was to quantitatively assess perchlorate concentrations in commercially available powdered infant formulas (PIFs). Secondary objectives were: (1) to estimate exposure in infants under different dosing scenarios and compare them with the perchlorate reference dose (RfD); (2) estimate the perchlorate concentration in water used for preparing PIFs that would result in a dose exceeding the RfD; and (3) estimate iodine intakes from PIFs. We quantified perchlorate levels in three samples (different lot numbers) of reconstituted PIF (using perchlorate-free water) from commercial brands of PIF in each of the following categories: bovine milk-based with lactose, soy-based, bovine milk-based but lactose-free, and elemental (typically consisting of synthetic amino acids). Exposure modeling was conducted to determine whether the RfD might be exceeded in 48 dosing scenarios that were dependent on age, centile energy intake per unit of body weight, body weight percentile, and PIF perchlorate concentration. We obtained three different samples in each of the five brands of bovine- and soy-based PIF, three different samples in each of the three brands of lactose-free PIF, and three different samples in two brands of elemental PIF. The results were as follows: bovine milk-based with lactose (1.72 microg/l, range: 0.68-5.05); soy-based (0.21 microg/l, range: 0.10-0.44); lactose-free (0.27 microg/l, range: 0.03-0.93); and elemental (0.18 microg/l, range: 0.08-0.4). Bovine milk-based PIFs with lactose had a significantly higher concentration of perchlorate (P<0.05) compared with all. Perchlorate was a contaminant of all commercially available PIFs tested. Bovine milk-based PIFs with lactose had a significantly higher perchlorate concentration perchlorate than soy, lactose-free, and elemental PIFs. The perchlorate RfD may be exceeded when certain bovine milk-based PIFs are ingested and/or when PIFs are reconstituted with perchlorate-contaminated water.
“Then the United States delegation, embracing the interests of infant formula manufacturers, upended the deliberations.
American officials sought to water down the resolution by removing language that called on governments to “protect, promote and support breast-feeding” and another passage that called on policymakers to restrict the promotion of food products that many experts say can have deleterious effects on young children.
When that failed, they turned to threats, according to diplomats and government officials who took part in the discussions. Ecuador, which had planned to introduce the measure, was the first to find itself in the cross hairs.
The Americans were blunt: If Ecuador refused to drop the resolution, Washington would unleash punishing trade measures and withdraw crucial military aid. The Ecuadorean government quickly acquiesced.”
(Surprised that The New York Times does not capitalize on this headline and include a formula advertisement.)
https://mobile.nytimes.com/2018/07/08/health/world-health-breastfeeding-ecuador-trump.html
The “Council of Gods” have literally destroyed millions of years of evolutionary development. Hormones in breast milk also build infant brains. Microbes ride the vagus nerve of infants to help develop their brains. Your hippocampus is loaded with estrogen receptors and develops from the hormones in breast milk.
“Seung’s new book, Connectome: How the Brain’s Wiring Makes Us Who We Are, explains how mapping out our neural connections in our brains might be the key to understanding the basis of things like personality, memory, perception and ideas, as well as illnesses that happen in the brain, like autism and schizophrenia.
“These kinds of disorders have been a puzzle for a long time,” says Seung. “We can look at other brain diseases, like Alzheimer’s disease and Parkinson’s disease, and see clear evidence that there is something wrong in the brain.”
But with schizophrenia and autism, there’s no clear abnormality during autopsy dissections, says Seung.
“We believe these are brain disorders because of lots of indirect evidence, but we can’t look at the brain directly and see something is wrong,” he says. “So the hypothesis is that the neurons are healthy, but they are simply connected together or organized in an abnormal way.”
One current theory, says Seung, is that there’s a connection between the wiring that develops between neurons during early infancy and developmental disorders like schizophrenia and autism.
“In autism, the development of the brain is hypothesized to go awry sometime before age 2, maybe in the womb,” he says. “In schizophrenia, no one knows for sure when the development is going off course. We know that schizophrenia tends to emerge in early adulthood, so many people believe that something abnormal is happening during adolescence. Or it could be that something is happening much earlier and it’s not revealed until you become an adult.”
What scientists do know, he says, is that the wiring of the brain in the first three years is critical for development. Infants born with cataracts in poor countries that don’t have the resources to restore their eyesight remain blind even after surgery is performed on them later in life.
“No matter how much they practice seeing, they can never really see,” says Seung. “They recover some visual function, but they are still blind by comparison to you and me. And one hypothesis is that the brain didn’t wire up properly when they were babies, so by the time they become adults, there’s no way for the brain to learn how to see properly.”
At birth, he says, you are born with all of the neurons you will ever have in life, except for neurons that exist in two specific areas of the brain: the dentate gyrus of the hippocampus, which is thought to help new memories form, and the olfactory bulb, which is involved in your sense of smell.
“The obvious hypothesis [is] that these two areas need to be highly plastic and need to learn more than other regions, and that’s why new neurons have to be created — to give these regions more potential for learning,” says Seung. “But we don’t really have any proof of that hypothesis.”
But not everything is set in stone from birth. The complex synaptic connections that allow neurons to communicate with one another develop after babies have left the womb.
“As far as we know, this is happening throughout your life,” he says. “Part of the reason that we are lifelong learners — that no matter how old you get, you can still learn something new — may be due to the fact that synapse creation and elimination are both continuing into adulthood.”
https://www.goodreads.com/book/show/11346470-connectome
NARRATOR: But, in comparing the brain scans of identical twins discordant for autism, Kaufman finally saw the definitive data he was searching for: an area in the brain linked to learning, memory and emotions—called the hippocampus—was smaller in the twin with severe autism. But how could the same genes create different brain structures? Kaufmann asked Andy Feinberg at Johns Hopkins University.
ANDREW FEINBERG: And suddenly we were able to form an epigenetic hypothesis. And that hypothesis is that they have the same genome, but one of them maybe has an epigenetic change that’s leading to a difference in their brain that you don’t see in the other twin.
NARRATOR: Kaufmann and Feinberg are now searching for methyl marks in the DNA of identical twins discordant for autism. The work has just begun, but the hope is that by finding identical genes that differ in their expression, some causes of autism may emerge.
Epigenetic changes occur from external or environmental impacts.
Ghost in Your Genes
https://www.pbs.org/wgbh/nova/transcripts/3413_genes.html
Organochlorine munitions in bomb, pill, spray, injection, and chemical additive forms shrink the hippocampus. The article “U.S. Nerve Gas Hit Our Own Troops in Iraq” explains, “According to Dr. Linda Chao, a neurologist at the University of California Medical School in San Francisco, “Because part of their brains, the hippocampus, has shrunk, they’re at greater risk for Alzheimer’s and other degenerative diseases.”
A Czech chemical-weapons detection unit found “trace concentrations of sarin, a nerve-paralyzing substance” drifting into Saudi Arabia. French, British and U.S. intelligence units found similar evidence.
Tracy Elledge, a former combat engineer and one of the veterans I interviewed, said, “Alarms went off all the time.… Our officers told us they were false and to disconnect them.”
However, Elledge and others were breathing poison.
https://renchemista.wordpress.com/2017/04/21/u-s-nerve-gas-hit-our-own-troops-in-iraq-by-barbara-koeppel/
Remember the “A” in Sarin stands for Nazi Otto Ambros and he expanded all organochlorine munition markets after serving only 3 years for his mass murder slavery conviction. He was IG Farben’s director of chemical operations and Hitler’s Director of Chemical Weapons. The Council of Gods were fully aware of the biological impacts of their organochlorine munitions and that was why they rounded up their infant and children victims in Vienna and exterminated them. They had market expansion plans so they had to cover up the damage.
Asperger’s Children: The Origins of Autism in Nazi Vienna by Edith Sheffer
https://renchemista.wordpress.com/2018/11/15/important-book-excerpts-from-aspergers-children-the-origins-of-autism-in-nazi-vienna-by-edith-sheffer-2/
Silent Spring by Rachel Carson
Excerpt From Chapter 13: Through a Narrow Window
Plants treated with benzene hexachloride (BHC) or lindane became monstrously deformed with tumorlike swellings on their roots. Their cells grew in size, being swollen with chromosomes which doubled in number. The doubling continued in future divisions until further cell division became mechanically impossible.
The herbicide 2,4-D has also produced tumor like swellings in treated plants. Chromosomes become short, thick, clumped together. Cell division is seriously retarded. The general effect is said to parallel closely that produced by X-rays.
Recent medical findings in the field of chromosome abnormalities are of extreme interest and significance. In 1959 several British and French research teams found their independent studies pointing to a common conclusion—that some of humanity’s ills are caused by a disturbance of the normal chromosome number. In certain diseases and abnormalities studied by these investigators the number differed from the normal. To illustrate: it is now known that all mongoloids have one extra chromosome. Occasionally this is attached to another so that the chromosome number remains the normal 46. As a rule, however, the extra is a separate chromosome, making the number 47. In such individuals, the original cause of the defect must have occurred in the generation preceding its appearance.
A different mechanism seems to operate in a number of patients, both in America and Great Britain, who are suffering from a chronic form of leukemia. These have been found to have a consistent chromosome abnormality in some of the blood cells. The abnormality consists of the loss of part of a chromosome. In these patients the skin cells have a normal complement of chromosomes. This indicates that the chromosome defect did not occur in the germ cells that gave rise to these individuals, but represents damage to particular cells (in this case, the precursors of blood cells) that occurred during the life of the individual. The loss of part of a chromosome has perhaps deprived these cells of their “instructions” for normal behavior.
The list of defects linked to chromosome disturbances has grown with surprising speed since the opening of this territory, hitherto beyond the boundaries of medical research. One, known only as Klinefelter’s syndrome, involves a duplication of one of the sex chromosomes. The resulting individual is a male, but because he carries two of the X chromosomes (becoming XXY instead of XY, the normal male complement) he is somewhat abnormal. Excessive height and mental defects often accompany the sterility caused by the condition. In contrast, an individual who receives only one sex chromosome (becoming XO instead of either XX or XY) is actually female but lacks many of the secondary sexual characteristics. The condition is accompanied by various physical (and sometimes mental) defects, for of course the X chromosome carries genes for a variety of characteristics. This is known as Turner’s syndrome…
An immense amount of work on the subject of chromosome abnormalities is being done by workers in many countries. A group at the University of Wisconsin, headed by Dr. Klaus Patau, has been concentrating on a variety of congenital abnormalities, usually including mental retardation, that seem to result from the duplication of only part of a chromosome, as if somewhere in the formation of one of the germ cells a chromosome had broken and the pieces had not been properly redistributed. Such a mishap is likely to interfere with the normal development of the embryo.
According to present knowledge, the occurrence of an entire extra body chromosome is usually lethal, preventing survival of the embryo. Only three such conditions are known to be viable; one of them, of course, is mongolism. The presence of an extra attached fragment, on the other hand, although seriously damaging is not necessarily fatal, and according to the Wisconsin investigators this situation may well account for a substantial part of the so far unexplained cases in which a child is born with multiple defects, usually including mental retardation.
This is so new a field of study that as yet scientists have been more concerned with identifying the chromosome abnormalities associated with disease and defective development than with speculating about the causes. It would be foolish to assume that any single agent is responsible for damaging the chromosomes or causing their erratic behavior during cell division. But can we afford to ignore the fact that we are now filling the environment with chemicals that have the power to strike directly at the chromosomes, affecting them in the precise ways that would cause such conditions? Is this not too high a price to pay for a sproutless potato or a mosquitoes patio?
We can, if we wish, reduce this threat to our genetic heritage, a possession that has come down to us through some two billion years of evolution and selection of living protoplasm, a possession that is ours for the moment only, until we must pass it on to the generations to come. We are doing little now to preserve its integrity. Although chemical manufacturers are required by law to test their materials for toxicity, they are not required to make the tests that would reliably demonstrate genetic effect, and they do not do so.
(That’s because manufacturers were already well aware of the biological impacts of the technologies they were expanding)